Din skjoldbruskkirtel spiller en væsentlig rolle i næsten alle metaboliske (biokemisk omsætning) processer, og når skjoldbruskkirtlen ikke fungerer, vil du ikke have det godt.
Har du svært ved at tabe dig, og er dit stofskifte for lavt. Undgå disse fødevarer
Lavt Stofskifte – HypoThyroidea – Hashimoto’s sygdom - alternativ behandling
Du ødelægger dit stofskifte hvis du sulter dig selv, eller følger en streng diæt. Har du nået et punkt, hvor du ikke længere kan tabe dig, skal du læse med her. Vi fortæller dig hvordan du kan
Er der virkelig mere end 300 symptomer på stofskifteproblemer? Hvem kunne vide, at en lille sommerfugleformet skjoldbruskkirtel i vores hals kan påvirke vores liv helt utroligt? Vi må ikke undervurdere den lille kirtels betydning. Den er chefen for kontrolcentret for de metaboliske funktioner i hver eneste celle i vores krop. Den har derfor beføjelse til at forstyrre alle dele af din krop og til at skabe dybtgående ændringer i alle aspekter af dit liv. Videnskabelig forskning linker hypothyroidisme (lavt stofskifte) til hjertesygdomme, diabetes, nyresygdomme, galdeblæren, leversygdomme, cancer og Alzheimers. Thyroid Federation International anslår i 2014, at der er op mod 750 millioner stofskiftesyge på verdensplan, som oplever dysfunktion i skjoldbruskkirtlen. Over halvdelen formodes at være uvidende om tilstanden. Trods forskning forbindes en underaktiv skjoldbruskkirtel ikke til nogen af de dødeligste sygdomme i vor tid. Hypothyroidisme epidemien fejer hen over hele vores klode og er stort set ukendt i det konventionelle medicinske samfund. Er du sikker på, at du er klar til denne lange, patetiske liste af symptomer? En læser kontaktede Dana Trentini med følgende spørgsmål. Spørgsmålet var så vigtigt, at det fik hende til at tænke, og tænke over, hvordan man bedst kunne løse dette. Kronisk urticaria (nældefeber) er en af de hundredvis af potentielle hypothyroidisme symptomer. Hej Dana – For 6 måneder siden, begyndte jeg at få nældefeber hver aften. Blodprøver og forskning på web har ikke givet nogen hjælp. Jeg læste noget info på din side, og jeg fandt en potentiel forbindelse mellem min tilstand og skjoldbruskkirtlen. Her en kort oversættelse fra siden: En stor procentdel af mennesker med kronisk urticaria eller nældefeber har en underliggende autoimmun proces som årsag til deres symptomer. Før i tiden blev en lille procentdel af mennesker med kronisk urticaria diagnosticeret med autoimmune sygdomme, såsom hypothyroidisme. Nogle af disse mennesker fik bedring, hvis deres sygdom i skjoldbruskkirtlen blev behandlet. En nylig undersøgelse af en gruppe allergiske patienter i Illinois (USA) fandt, at en større procentdel af mennesker med kronisk urticaria har stofskiftesygdomme end tidligere troet – op til 20%. Kilde: allergies.about.com Så hvis du har kronisk nældefeber, skulle du få undersøgt, om en lav skjoldbruskkirtel funktion er årsagen til dine symptomer. Mit håb er, at denne artikel finder de millioner af mennesker verden over, der ikke er klar over deres tilstand. De lider af flere sundhedsmæssige komplikationer, men de har ingen anelse om, at deres skjoldbruskkirtel har skylden. Deres læger ordinerer medicin efter medicin til sygdomme, der kunne behandles ved at behandle den underliggende hypothyroidisme. Det er en tragedie. Så er der dem, der har været diagnosticeret med hypothyroidisme og behandles med konventionel medicin, mens de stadig lider af mange symptomer på denne liste. Deres læger har ingen idé om, at den egentlige årsag til deres sygdom er forkert behandlet hypothyroidisme. De bliver ved med at ordinere flere medikamenter til sygdomme, der udmærket kan behandles ved korrekt medicinering. Andre bliver fejlagtigt betragtet som hypokondere og får ordineret sovepiller og antidepressiv medicin af deres læger. Andre igen får at vide, at deres symptomer blot er en del af den normale aldring og affærdiges som uvæsentlig. Mange læger stoler udelukkende på laboratorieresultater til diagnose, ikke på fuldt ud at undersøge patientens sygehistorie, symptomer, familie historie og grundige fysiske undersøgelser. Lyder det som dig? Det kan være tid til at skifte læge. Følgende personer og organisationer er nogle af de øverste sundhedseksperter i verden. Dana’s liste over 300 + symptomer er en samling fra deres lister over hypothyreose symptomer, sammen med yderligere symptomer jeg har fundet i mine undersøgelser. Tak til Dr. Mark Starr for omfattende liste over hypothyroidisme symptomer i sin bog ”Hypothyroidisme Type 2: Epidemien”. Tak til stofskifte pioneren Dr. Broda Barnes for hans grundige beskrivelse af symptomer i den legendariske bog ”Hypothyroidisme: Den upåagtede Sygdom”. Tak advokat Mary Shomon for hendes omfattende liste over hypothyroidisme symptomer i hendes bøger” Living Well with Hypothyroidism: “What Your Doctor Doesn’t Tell You” “That You Need to Know” og “The Thyroid Hormone Breakthrough”. Tak også til ThyroidUK for at levere mange ressourcer. Mennesker kan have forskellige kombinationer af symptomer. Sværhedsgraden af hver persons hypothyroidisme vil også bestemme sværhedsgraden og antallet af symptomer, de oplever. Ubehandlet hypothyroidisme forværres med alderen med forværrede symptomer. Du må ikke forledes til at tro, at symptomer fra denne liste ikke er forbundet til hypothyroidisme, fordi det kan de sagtens være i dit tilfælde, selvom din læge ikke kender til dem. Lider du af nogle af disse symptomer? Har du fået ordineret medicin for symptomer som anført nedenfor? Er du sikker på din læge er opmærksom på forbindelsen af disse symptomer til hypothyroidisme? Er du i behandling med konventionel medicin, og har du stadig symptomer? Er der sygdom i din familie, i skjoldbruskkirtlen, hjertesygdomme, diabetes, autoimmune sygdomme, kræft og/eller Alzheimers? Den lange liste over hypothyroidisme symptomer En utrolig lang liste. Var du klar over det? Kilde http://hypothyroidmom.com Frit oversat af Anett Kromann med tilladelse fra Dana Trentini Opdateret 26.01.2016
Thyroid hormones are responsible for metabolizing fat and carbs; they regulate respiration, cholesterol levels, heart function, body…
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Your thyroid is the single most important gland in your body because it plays a role in regulating all other hormones and systems. It also happens to be incredibly sensitive to common problems like stress,
The thyroid and nasal cavity connection has been well known for a century. The runny nose, postnasal drip, cough, congestion, loss of smell, chronic sinus infections, and sinus headaches of hypothyroidism all make perfect sense.
I've taken a bit of time away from blogging because I've been swamped with clients and wanted to spend what free time I have reading. I've come across some very interesting stuff that I believe plays a significant role in hormonal balance that no one is covering so I'll do my best to break it down in to digestible information. Anabolic carbohydrate metabolism When people think of carbohydrate metabolism, they immediately think of glycolysis and maybe even the citric acid cycle. Both of these reactions are catabolic, they are oxidative processes that make ATP through the breakdown of glucose. However, there is another pathway of glucose metabolism called the pentose phosphate pathway that is anabolic in nature. The function of the pentose phosphate pathway is to produce reducing equivalents to power anabolic reactions, to produce ribose 5 phosphate(R5P) for the synthesis of DNA, and to help shuffle carbons between sugars so that they can be utilized again. But why is this important? Let's take a look. Anaerobic glycolysis is the breakdown of glucose in the cytosol of cells. The first step uses ATP to make glucose 6 phosphate(G6P) which can either continue along the pathway of glycolysis to make ATP or be directed through the pentose phosphate pathway to produce reducing equivalents and/or R5P. The path that G6P takes is ultimately dictated by cellular needs. To make this uber-confusing I'll post the steps of both pathways below. It's not really important that you focus on the steps so much as the parallel pathways that glucose metabolism can take based on cellular needs. Depending on cell type, there are different levels of pentose phosphate pathway activity. Cells that are exposed to high levels of oxidative stress, that produce hormones, and that have a high turnover rate tend to have more flux through the pentose phosphate pathway. But why? The 2 phases of the pentose phosphate pathway There are 2 phases of the pentose phosphate pathway, an oxidative phase and a non-oxidative phase, both shown below. The oxidative phase of the pentose phosphate pathway The oxidative phase reduces 2 nicotinamide adenine dinucleuotide phosphates (NADP+) to 2 NADPH, the reduced form. This is important for 2 reasons. First, NADPH is needed to convert oxidized glutathione to its reduced form. Everyone and their mother who is trying to optimize health is likely taking some form of supplement or eating foods that promote glutathione production. Glutathione is known as the master antioxidant as it goes around donating electrons to free radicals before they can react with cellular structures. The problem is, once glutathione does this it's converted to its oxidized form which can no longer donate electrons. In this form, glutathione is essentially worthless until NADPH reduces it back to the active form. This converts NADPH back to NADP+ where the oxidative phase of the pentose phosphate pathway can swing back in and produce more NADPH. For this reason, any cell that is subject to high levels of oxidative stress such as red blood cells and liver cells has a very active pentose phopshate pathway. The figure below illustrates the process by which NADPH reduces oxidized glutathione. Note that selenium is needed in order for glutathione to reduce free radicals and NADPH and riboflavin are needed to reduce oxidized glutathione back to its active form. Without NADPH, glutathione remains inactive, something you most certainly do not want. The pentose phosphate pathway is the primary way in which our cells make NADPH for this purpose. In addition, cells of the immune system used NADPH to kill foreign pathogens through a process called the respiratory burst. A decrease in the ratio of NADPH:NADP+ ratio will compromise the immune system and provide an environment where pathogens can evade killing by phagocytosis. The next reason that the oxidative phase is important is because reducing power is needed for all anabolic processes. This includes the biosynthesis of hormones, so there are high levels of pentose phosphate activity in the adrenal and sex glands. With insufficient reducing power in the form of NADPH, hormonal balance can be thrown out of whack. This includes the adrenal and sex hormones as well as the the thyroid, but for a different reason. Several reversible hormonal conversions are dependent on the ratio of NADPH to NADP+ including: DHEA Androstenediol Androstenedione Testosterone Estrone Estradiol Cortisone Cortisol NADPH causes the conversions to go from left to right while NADP+ causes them to go from right to left. So what does this mean? NADPH will convert androstenedione to testosterone while NADP+ will convert testosterone to androstenedione. In other words, reduced flux through the oxidative arm of the pentose phosphate pathway will negatively impact testosterone levels in men because there will be more NADP+ than NADPH which favors androstenedione over testosterone. I am no expert on women's hormonal issues, but given the fact that estrone is the predominant form of estrogen in postmenopausal women while estradiol is the predominant form in women of reproductive age, I'll go out on a limb and say this conversion is likely detrimental. On the adrenal side of things, reduced flux through the oxidative arm of the pentose phosphate pathway will negatively impact cortisol levels by favoring cortisone production. Since cortisol has greater glucocorticoid activity and cortisone has no mineralocorticoid activity, this can have a pretty significant effect on glucose levels, inflammation, and electrolyte balance. Since these hormonal conversions occur in the endoplasmic reticulum(ER) of cells, the ratio of NADPH:NADP+ within the ER can have a pretty significant effect on hormonal balance. Reduced flux through the pentose phosphate pathway and high levels of oxidative stress are 2 factors that can decrease this ratio leading to poor hormonal balance. With the thyroid, the proposed mechanism by which reduced flux through the oxidative arm of the pentose phosphate pathway may negatively impact hormone production is through a reduction in glutathione function. The thyroid relies on hydrogen peroxide, a free radical, for thyroid hormone synthesis. Without sufficient levels of reduced glutathione to keep this in check, the thyroid can become damaged, inflamed, and thyroid function can become compromised. The non-oxidative phase of the pentose phosphate pathway The non-oxidative arm of the pentose phosphate pathway is also important, especially in tissues with high rates of cell turnover. The non-oxidative arm of the pentose phosphate pathway essentially interconverts sugars in to different forms. One of those sugars, R5P, is necessary for DNA production. Cells with high turnover rates such as epithelial cells in the gut are dependent on sufficient flux through the non-oxidative arm to produce enough DNA for replication. The non-oxidative arm also gives the pentose phosphate pathway flexibility. By interconverting sugars, the non-oxidative arm can create sugars that can re-enter glycolysis to generate pyruvate or feed back in to the oxidative arm to create more NADPH. The non-oxidative arm allows the pentose phosphate pathway and glycolytic pathways to work synergistically to meet cell needs. There are essentially 4 modes of pentose phosphate pathway activity that can be used to increase energy levels, increase reducing power, provide building blocks for DNA, or a combination of these functions. The yield of each mode is: Mode 1-6 G6P makes 5 R5P Mode 2-1 G6P makes 1 R5P and 2 NADPH Mode 3 -1 G6P makes 12 NADPH Mode 4-3 G6P make 6 NADPH, 8 ATP, & 5 pyruvate and NADH which can be used to create more ATP As you can see, carbohydrate metabolism is a lot more than simply breaking down glucose to use for energy. Carbohydrate metabolism also has anabolic effects via the pentose phosphate pathway. Decreased flux through the pentose phosphate pathway can increase oxidative stress and negatively impact hormonal balance and cellular reproduction. There is compelling scientific evidence that altered flux through the pentose phosphate pathway may be at the root of adrenal dysfunction, increased inflammation, and some gut pathologies including SIBO. In addition, many metabolic consequences of Type 2 diabetes can alter flux through the pentose phosphate pathway and there are many blood markers we see in Type 2 diabetes that indicate this. If there is enough demand I may pull some of that stuff out, so read, like, and share.
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If you are feeling something strange in your body it could be a subtle sign that something is amiss with your thyroid so pay attention.
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L'hyperthyroïdie chez les femmes est une maladie commune qui affecte le système métabolique. Elle se caractérise par une production anormale…
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